User:User123: Difference between revisions
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https://www.nature.com/articles/s41422-021-00495-9 - SARS COV 2 spike protein activates TLR4 and acts as a superantigen | https://www.nature.com/articles/s41422-021-00495-9 - SARS COV 2 spike protein activates TLR4 and acts as a superantigen | ||
=== TLR4 === | |||
https://pubmed.ncbi.nlm.nih.gov/18203139/ - !!! The TLR4 pathway seems to regulate the priming of Th17 cells and reduce the severity of EAE. | https://pubmed.ncbi.nlm.nih.gov/18203139/ - !!! The TLR4 pathway seems to regulate the priming of Th17 cells and reduce the severity of EAE. | ||
https://pubmed.ncbi.nlm.nih.gov/21685327/ - This study had conflicting results. TLR4 knockout mice had the same susceptibility to disease. | |||
Revision as of 03:51, 9 September 2022
Stuff I'm working on
Post_vaccination_syndrome_introduction_for_practitioners
https://docs.google.com/document/d/1UgVw3wa43hzORX4wzCDfLldfiO0D_bRcD1G_HJFq0bQ/edit?usp=sharing - A theory regarding the cause of vaccine injury
Super antigen theory notes
https://sci-hub.se/https://doi.org/10.1016/0882-4010(87)90003-9 - In the TMEV virus, inbred mice can be susceptible or resistant to the virus.
https://www.nature.com/articles/s41422-021-00495-9 - SARS COV 2 spike protein activates TLR4 and acts as a superantigen
TLR4
https://pubmed.ncbi.nlm.nih.gov/18203139/ - !!! The TLR4 pathway seems to regulate the priming of Th17 cells and reduce the severity of EAE.
https://pubmed.ncbi.nlm.nih.gov/21685327/ - This study had conflicting results. TLR4 knockout mice had the same susceptibility to disease.